File:The impact of lymphangiogenesis in transplant arteriosclerosis.png

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Description Figure 5. The impact of lymphangiogenesis in transplant arteriosclerosis. After vascular transplantation, the immediate recognition of donor artery components progressively induces immunological rejection response. After injecting a highly selective inhibitor for VEGFR3, the inhibition of lymphangiogenesis effectively suppresses the cellular adaptive immunity, and further ameliorates neointimal hyperplasia, adventitial fibrosis, and teritary lymphoid organs’ (TLOs) formation in vascular allografts. Resultantly, an immunosuppressive circle was reasonably achieved by the negative lymphangiogenesis manipulating. LEC: lymphatic endothelial cell.
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Source https://www.mdpi.com/cells/cells-11-04056/article_deploy/html/images/cells-11-04056-g003.png https://doi.org/10.3390/cells11244056 The Impact of Stem/Progenitor Cells on Lymphangiogenesis in Vascular Disease. Cells 2022, 11, 4056.
Author Mou, R.; Chen, K.; Zhu, P.; Xu, Q.; Ma, L.
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© 2022 by the authors. Licensee MDPI, Basel, Switzerland. This article is an open access article distributed under the terms and conditions of the Creative Commons Attribution (CC BY) license (https://creativecommons.org/licenses/by/4.0/).

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[[Category:Lymphangiogenesis]

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current17:02, 26 April 2024Thumbnail for version as of 17:02, 26 April 20243,271 × 1,615 (1.25 MB)Rasbak (talk | contribs){{Information |description=Figure 5. The impact of lymphangiogenesis in transplant arteriosclerosis. After vascular transplantation, the immediate recognition of donor artery components progressively induces immunological rejection response. After injecting a highly selective inhibitor for VEGFR3, the inhibition of lymphangiogenesis effectively suppresses the cellular adaptive immunity, and further ameliorates neointimal hyperplasia, adventitial fibrosis, and teritary lymphoid organs’ (TLOs)...

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