File:APP and LTP in Alzheimer disease.png: Difference between revisions

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== [[Commons:Copyright tags|Licensing]]: ==
== [[Commons:Copyright tags|Licensing]]: ==
{{PD-self}}
{{PD-self}}
{{Uncategorized|year=2008|month=November|day=10}}
{{Check categories|year=2008|month=November|day=10
|lang1=en|wiki1=wikipedia|article1=Long-term_potentiation
}}
[[Category:Long-term potentiation]]

Revision as of 16:48, 10 November 2008

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English: Rowen et al's proposal for how misprocessing of amyloid precursor protein (APP) leads to decreased long-term potentiation (LTP) and early and late cognitive decline in individuals with Alzheimer disease. From the article: Rowan MJ, Klyubin I, Cullen WK, Anwyl R (April 2003). "Synaptic plasticity in animal models of early Alzheimer's disease". Philosophical transactions of the Royal Society of London. Series B, Biological sciences 358 (1432): 821–8. doi:10.1098/rstb.2002.1240. PMID 12740129. PMC: 1693153, http://journals.royalsociety.org/openurl.asp?genre=article&issn=0962-8436&volume=358&issue=1432&spage=821.
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current14:52, 9 November 2008Thumbnail for version as of 14:52, 9 November 2008239 × 322 (23 KB)Diberri (talk | contribs){{Information |Description={{en|1=Rowen ''et al'''s proposal for how misprocessing of amyloid precursor protein (APP) leads to decreased long-term potentiation (LTP) and early and late cognitive decline in individuals with Alzheimer disease. From the arti

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