Fixed the whole article. Please read Talk Page before changing the terms. Respiratory compensation is ONLY for metabolic imbalances, NOT for respiratory imbalances.
'''Respiratory compensation''' is a mechanism of the [[respiratory center]] by which involves altering [[alveolar ventilation]] to try and bring the plasma [[pH]] canback beto alteredits bynormal varyingvalue (7.4) in order to keep the [[respiratoryAcid–base ratehomeostasis|acid-base balance]] in the body. It usually occurs within minutes to hours and is much faster than [[renal compensation]] (which takes several days), but has less ability to restore normal values.
In [[respiratorymetabolic alkalosisacidosis]], [[chemoreceptors]] sense a deranged [[Acid–base homeostasis|acid-base systembalance]] with a plasma [[pH]] of less than normal (<7.4). The chemoreceptors send afferent fibers to the brainstem respiratory centers. The [[Respiratory center|brainstem respiratory centers]] increase alveolar ventilation ([[hyperventilation]]) so that [[carbon dioxide]] (CO2) can be breathed off, andresulting therein isan increasedincrease breathingof plasma pH.<ref>{{GeorgiaPhysiology|7https://www.ncbi.nlm.nih.gov/books/7ch12NBK507807/7ch12p53}}</ref> The amount of respiratory compensation in metabolic acidosis can be estimated using [[Winters' formula]].
In [[metabolic alkalosis]], [[chemoreceptors]] sense a deranged [[Acid–base homeostasis|acid-base balance]] with a plasma [[pH]] of more than normal (>7.4). The chemoreceptors send afferent fibers to the brainstem respiratory centers. The [[Respiratory center|brainstem respiratory centers]] decrease alveolar ventilation ([[hypoventilation]]) to create a rise in arterial [[carbon dioxide]] (CO2) tension, resulting in a decrease of plasma pH.<ref>https://www.ncbi.nlm.nih.gov/books/NBK507807/</ref>
In [[respiratory acidosis]], the breathing rate is decreased.<ref>{{GeorgiaPhysiology|7/7ch12/7ch12p58}}</ref>