ARID3A: Difference between revisions
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*{{cite journal | author=Kortschak RD, Tucker PW, Saint R |title=ARID proteins come in from the desert. |journal=Trends Biochem. Sci. |volume=25 |issue= 6 |pages= |
*{{cite journal | author=Kortschak RD, Tucker PW, Saint R |title=ARID proteins come in from the desert. |journal=Trends Biochem. Sci. |volume=25 |issue= 6 |pages= 294–9 |year= 2000 |pmid= 10838570 |doi= }} |
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*{{cite journal | author=Kortschak RD, Reimann H, Zimmer M, ''et al.'' |title=The human dead ringer/bright homolog, DRIL1: cDNA cloning, gene structure, and mapping to D19S886, a marker on 19p13.3 that is strictly linked to the Peutz-Jeghers syndrome. |journal=Genomics |volume=51 |issue= 2 |pages= |
*{{cite journal | author=Kortschak RD, Reimann H, Zimmer M, ''et al.'' |title=The human dead ringer/bright homolog, DRIL1: cDNA cloning, gene structure, and mapping to D19S886, a marker on 19p13.3 that is strictly linked to the Peutz-Jeghers syndrome. |journal=Genomics |volume=51 |issue= 2 |pages= 288–92 |year= 1998 |pmid= 9722953 |doi= 10.1006/geno.1998.5259 }} |
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*{{cite journal | author=Suzuki M, Okuyama S, Okamoto S, ''et al.'' |title=A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer. |journal=Oncogene |volume=17 |issue= 7 |pages= |
*{{cite journal | author=Suzuki M, Okuyama S, Okamoto S, ''et al.'' |title=A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer. |journal=Oncogene |volume=17 |issue= 7 |pages= 853–65 |year= 1998 |pmid= 9780002 |doi= 10.1038/sj.onc.1202163 }} |
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*{{cite journal | author=Peeper DS, Shvarts A, Brummelkamp T, ''et al.'' |title=A functional screen identifies hDRIL1 as an oncogene that rescues RAS-induced senescence. |journal=Nat. Cell Biol. |volume=4 |issue= 2 |pages= |
*{{cite journal | author=Peeper DS, Shvarts A, Brummelkamp T, ''et al.'' |title=A functional screen identifies hDRIL1 as an oncogene that rescues RAS-induced senescence. |journal=Nat. Cell Biol. |volume=4 |issue= 2 |pages= 148–53 |year= 2002 |pmid= 11812999 |doi= 10.1038/ncb742 }} |
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*{{cite journal | author=Kaiwen M |title=[Involvement of E2FBP1, an ARID family member protein, in the p53 regulatory pathway] |journal=Kokubyo Gakkai Zasshi |volume=69 |issue= 2 |pages= |
*{{cite journal | author=Kaiwen M |title=[Involvement of E2FBP1, an ARID family member protein, in the p53 regulatory pathway] |journal=Kokubyo Gakkai Zasshi |volume=69 |issue= 2 |pages= 152–61 |year= 2002 |pmid= 12136662 |doi= }} |
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*{{cite journal | author=Goebel P, Montalbano A, Ayers N, ''et al.'' |title=High frequency of matrix attachment regions and cut-like protein x/CCAAT-displacement protein and B cell regulator of IgH transcription binding sites flanking Ig V region genes. |journal=J. Immunol. |volume=169 |issue= 5 |pages= |
*{{cite journal | author=Goebel P, Montalbano A, Ayers N, ''et al.'' |title=High frequency of matrix attachment regions and cut-like protein x/CCAAT-displacement protein and B cell regulator of IgH transcription binding sites flanking Ig V region genes. |journal=J. Immunol. |volume=169 |issue= 5 |pages= 2477–87 |year= 2002 |pmid= 12193717 |doi= }} |
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*{{cite journal | author=Strausberg RL, Feingold EA, Grouse LH, ''et al.'' |title=Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences. |journal=Proc. Natl. Acad. Sci. U.S.A. |volume=99 |issue= 26 |pages= |
*{{cite journal | author=Strausberg RL, Feingold EA, Grouse LH, ''et al.'' |title=Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences. |journal=Proc. Natl. Acad. Sci. U.S.A. |volume=99 |issue= 26 |pages= 16899–903 |year= 2003 |pmid= 12477932 |doi= 10.1073/pnas.242603899 }} |
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*{{cite journal | author=Ma K, Araki K, Ichwan SJ, ''et al.'' |title=E2FBP1/DRIL1, an AT-rich interaction domain-family transcription factor, is regulated by p53. |journal=Mol. Cancer Res. |volume=1 |issue= 6 |pages= |
*{{cite journal | author=Ma K, Araki K, Ichwan SJ, ''et al.'' |title=E2FBP1/DRIL1, an AT-rich interaction domain-family transcription factor, is regulated by p53. |journal=Mol. Cancer Res. |volume=1 |issue= 6 |pages= 438–44 |year= 2004 |pmid= 12692263 |doi= }} |
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*{{cite journal | author=Fukuyo Y, Mogi K, Tsunematsu Y, Nakajima T |title=E2FBP1/hDril1 modulates cell growth through downregulation of promyelocytic leukemia bodies. |journal=Cell Death Differ. |volume=11 |issue= 7 |pages= |
*{{cite journal | author=Fukuyo Y, Mogi K, Tsunematsu Y, Nakajima T |title=E2FBP1/hDril1 modulates cell growth through downregulation of promyelocytic leukemia bodies. |journal=Cell Death Differ. |volume=11 |issue= 7 |pages= 747–59 |year= 2005 |pmid= 15017387 |doi= 10.1038/sj.cdd.4401412 }} |
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*{{cite journal | author=Grimwood J, Gordon LA, Olsen A, ''et al.'' |title=The DNA sequence and biology of human chromosome 19. |journal=Nature |volume=428 |issue= 6982 |pages= |
*{{cite journal | author=Grimwood J, Gordon LA, Olsen A, ''et al.'' |title=The DNA sequence and biology of human chromosome 19. |journal=Nature |volume=428 |issue= 6982 |pages= 529–35 |year= 2004 |pmid= 15057824 |doi= 10.1038/nature02399 }} |
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*{{cite journal | author=Nixon JC, Rajaiya JB, Ayers N, ''et al.'' |title=The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations. |journal=Cell. Immunol. |volume=228 |issue= 1 |pages= |
*{{cite journal | author=Nixon JC, Rajaiya JB, Ayers N, ''et al.'' |title=The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations. |journal=Cell. Immunol. |volume=228 |issue= 1 |pages= 42–53 |year= 2004 |pmid= 15203319 |doi= 10.1016/j.cellimm.2004.03.004 }} |
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*{{cite journal | author=Rajaiya J, Nixon JC, Ayers N, ''et al.'' |title=Induction of immunoglobulin heavy-chain transcription through the transcription factor Bright requires TFII-I. |journal=Mol. Cell. Biol. |volume=26 |issue= 12 |pages= |
*{{cite journal | author=Rajaiya J, Nixon JC, Ayers N, ''et al.'' |title=Induction of immunoglobulin heavy-chain transcription through the transcription factor Bright requires TFII-I. |journal=Mol. Cell. Biol. |volume=26 |issue= 12 |pages= 4758–68 |year= 2006 |pmid= 16738337 |doi= 10.1128/MCB.02009-05 }} |
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*{{cite journal | author=Olsen JV, Blagoev B, Gnad F, ''et al.'' |title=Global, in vivo, and site-specific phosphorylation dynamics in signaling networks. |journal=Cell |volume=127 |issue= 3 |pages= |
*{{cite journal | author=Olsen JV, Blagoev B, Gnad F, ''et al.'' |title=Global, in vivo, and site-specific phosphorylation dynamics in signaling networks. |journal=Cell |volume=127 |issue= 3 |pages= 635–48 |year= 2006 |pmid= 17081983 |doi= 10.1016/j.cell.2006.09.026 }} |
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*{{cite journal | author=Lin D, Ippolito GC, Zong RT, ''et al.'' |title=Bright/ARID3A contributes to chromatin accessibility of the immunoglobulin heavy chain enhancer. |journal=Mol. Cancer |volume=6 |issue= |pages= 23 |year= 2007 |pmid= 17386101 |doi= 10.1186/1476-4598-6-23 }} |
*{{cite journal | author=Lin D, Ippolito GC, Zong RT, ''et al.'' |title=Bright/ARID3A contributes to chromatin accessibility of the immunoglobulin heavy chain enhancer. |journal=Mol. Cancer |volume=6 |issue= |pages= 23 |year= 2007 |pmid= 17386101 |doi= 10.1186/1476-4598-6-23 }} |
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Revision as of 10:47, 11 June 2008
AT rich interactive domain 3A (BRIGHT-like), also known as ARID3A, is a human gene.[5]
References
- ^ a b c GRCh38: Ensembl release 89: ENSG00000116017 – Ensembl, May 2017
- ^ a b c GRCm38: Ensembl release 89: ENSMUSG00000019564 – Ensembl, May 2017
- ^ "Human PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
- ^ "Mouse PubMed Reference:". National Center for Biotechnology Information, U.S. National Library of Medicine.
- ^ "Entrez Gene: ARID3A AT rich interactive domain 3A (BRIGHT-like)".
Further reading
External links
- ARID3A+protein,+human at the U.S. National Library of Medicine Medical Subject Headings (MeSH)
This article incorporates text from the United States National Library of Medicine, which is in the public domain.