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==Interactions==
==Interactions==
ARID3A has been shown to [[Protein-protein_interaction|interact]] with [[E2F1]]<ref name=pmid9780002>{{cite journal | quotes = yes |last=Suzuki |first=M |authorlink= |coauthors=Okuyama S, Okamoto S, Shirasuna K, Nakajima T, Hachiya T, Nojima H, Sekiya S, Oda K |year=[[1998]]|month=Aug. |title=A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer |journal=Oncogene |volume=17 |issue=7 |pages=853–65 |publisher= |location = ENGLAND| issn = 0950-9232| pmid = 9780002 |doi = 10.1038/sj.onc.1202163 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = }}</ref> and [[Bruton's tyrosine kinase]].<ref name=pmid15203319>{{cite journal | quotes = yes |last=Nixon |first=Jamee C |authorlink= |coauthors=Rajaiya Jaya B, Ayers Neil, Evetts Seth, Webb Carol F |year=[[2004]]|month=Mar. |title=The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations |journal=Cell. Immunol. |volume=228 |issue=1 |pages=42–53 |publisher= |location = United States| issn = 0008-8749| pmid = 15203319 |doi = 10.1016/j.cellimm.2004.03.004 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = }}</ref>
ARID3A has been shown to [[Protein-protein_interaction|interact]] with [[E2F1]]<ref name=pmid9780002>{{cite journal |last=Suzuki |first=M |authorlink= |coauthors=Okuyama S, Okamoto S, Shirasuna K, Nakajima T, Hachiya T, Nojima H, Sekiya S, Oda K |year=[[1998]]|month=Aug. |title=A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer |journal=Oncogene |volume=17 |issue=7 |pages=853–65 |publisher= |location = ENGLAND| issn = 0950-9232| pmid = 9780002 |doi = 10.1038/sj.onc.1202163 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = }}</ref> and [[Bruton's tyrosine kinase]].<ref name=pmid15203319>{{cite journal |last=Nixon |first=Jamee C |authorlink= |coauthors=Rajaiya Jaya B, Ayers Neil, Evetts Seth, Webb Carol F |year=[[2004]]|month=Mar. |title=The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations |journal=Cell. Immunol. |volume=228 |issue=1 |pages=42–53 |publisher= |location = United States| issn = 0008-8749| pmid = 15203319 |doi = 10.1016/j.cellimm.2004.03.004 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = }}</ref>


==References==
==References==
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*{{cite journal | author=Kaiwen M |title=[Involvement of E2FBP1, an ARID family member protein, in the p53 regulatory pathway] |journal=Kokubyo Gakkai Zasshi |volume=69 |issue= 2 |pages= 152–61 |year= 2002 |pmid= 12136662 |doi= }}
*{{cite journal | author=Kaiwen M |title=[Involvement of E2FBP1, an ARID family member protein, in the p53 regulatory pathway] |journal=Kokubyo Gakkai Zasshi |volume=69 |issue= 2 |pages= 152–61 |year= 2002 |pmid= 12136662 |doi= }}
*{{cite journal | author=Goebel P, Montalbano A, Ayers N, ''et al.'' |title=High frequency of matrix attachment regions and cut-like protein x/CCAAT-displacement protein and B cell regulator of IgH transcription binding sites flanking Ig V region genes. |journal=J. Immunol. |volume=169 |issue= 5 |pages= 2477–87 |year= 2002 |pmid= 12193717 |doi= }}
*{{cite journal | author=Goebel P, Montalbano A, Ayers N, ''et al.'' |title=High frequency of matrix attachment regions and cut-like protein x/CCAAT-displacement protein and B cell regulator of IgH transcription binding sites flanking Ig V region genes. |journal=J. Immunol. |volume=169 |issue= 5 |pages= 2477–87 |year= 2002 |pmid= 12193717 |doi= }}
*{{cite journal | author=Strausberg RL, Feingold EA, Grouse LH, ''et al.'' |title=Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences. |journal=Proc. Natl. Acad. Sci. U.S.A. |volume=99 |issue= 26 |pages= 16899–903 |year= 2003 |pmid= 12477932 |doi= 10.1073/pnas.242603899 }}
*{{cite journal | author=Strausberg RL, Feingold EA, Grouse LH, ''et al.'' |title=Generation and initial analysis of more than 15,000 full-length human and mouse cDNA sequences. |journal=Proc. Natl. Acad. Sci. U.S.A. |volume=99 |issue= 26 |pages= 16899–903 |year= 2003 |pmid= 12477932 |doi= 10.1073/pnas.242603899 | pmc=139241 }}
*{{cite journal | author=Ma K, Araki K, Ichwan SJ, ''et al.'' |title=E2FBP1/DRIL1, an AT-rich interaction domain-family transcription factor, is regulated by p53. |journal=Mol. Cancer Res. |volume=1 |issue= 6 |pages= 438–44 |year= 2004 |pmid= 12692263 |doi= }}
*{{cite journal | author=Ma K, Araki K, Ichwan SJ, ''et al.'' |title=E2FBP1/DRIL1, an AT-rich interaction domain-family transcription factor, is regulated by p53. |journal=Mol. Cancer Res. |volume=1 |issue= 6 |pages= 438–44 |year= 2004 |pmid= 12692263 |doi= }}
*{{cite journal | author=Fukuyo Y, Mogi K, Tsunematsu Y, Nakajima T |title=E2FBP1/hDril1 modulates cell growth through downregulation of promyelocytic leukemia bodies. |journal=Cell Death Differ. |volume=11 |issue= 7 |pages= 747–59 |year= 2005 |pmid= 15017387 |doi= 10.1038/sj.cdd.4401412 }}
*{{cite journal | author=Fukuyo Y, Mogi K, Tsunematsu Y, Nakajima T |title=E2FBP1/hDril1 modulates cell growth through downregulation of promyelocytic leukemia bodies. |journal=Cell Death Differ. |volume=11 |issue= 7 |pages= 747–59 |year= 2005 |pmid= 15017387 |doi= 10.1038/sj.cdd.4401412 }}
*{{cite journal | author=Grimwood J, Gordon LA, Olsen A, ''et al.'' |title=The DNA sequence and biology of human chromosome 19. |journal=Nature |volume=428 |issue= 6982 |pages= 529–35 |year= 2004 |pmid= 15057824 |doi= 10.1038/nature02399 }}
*{{cite journal | author=Grimwood J, Gordon LA, Olsen A, ''et al.'' |title=The DNA sequence and biology of human chromosome 19. |journal=Nature |volume=428 |issue= 6982 |pages= 529–35 |year= 2004 |pmid= 15057824 |doi= 10.1038/nature02399 }}
*{{cite journal | author=Nixon JC, Rajaiya JB, Ayers N, ''et al.'' |title=The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations. |journal=Cell. Immunol. |volume=228 |issue= 1 |pages= 42–53 |year= 2004 |pmid= 15203319 |doi= 10.1016/j.cellimm.2004.03.004 }}
*{{cite journal | author=Nixon JC, Rajaiya JB, Ayers N, ''et al.'' |title=The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations. |journal=Cell. Immunol. |volume=228 |issue= 1 |pages= 42–53 |year= 2004 |pmid= 15203319 |doi= 10.1016/j.cellimm.2004.03.004 }}
*{{cite journal | author=Rajaiya J, Nixon JC, Ayers N, ''et al.'' |title=Induction of immunoglobulin heavy-chain transcription through the transcription factor Bright requires TFII-I. |journal=Mol. Cell. Biol. |volume=26 |issue= 12 |pages= 4758–68 |year= 2006 |pmid= 16738337 |doi= 10.1128/MCB.02009-05 }}
*{{cite journal | author=Rajaiya J, Nixon JC, Ayers N, ''et al.'' |title=Induction of immunoglobulin heavy-chain transcription through the transcription factor Bright requires TFII-I. |journal=Mol. Cell. Biol. |volume=26 |issue= 12 |pages= 4758–68 |year= 2006 |pmid= 16738337 |doi= 10.1128/MCB.02009-05 | pmc=1489113 }}
*{{cite journal | author=Olsen JV, Blagoev B, Gnad F, ''et al.'' |title=Global, in vivo, and site-specific phosphorylation dynamics in signaling networks. |journal=Cell |volume=127 |issue= 3 |pages= 635–48 |year= 2006 |pmid= 17081983 |doi= 10.1016/j.cell.2006.09.026 }}
*{{cite journal | author=Olsen JV, Blagoev B, Gnad F, ''et al.'' |title=Global, in vivo, and site-specific phosphorylation dynamics in signaling networks. |journal=Cell |volume=127 |issue= 3 |pages= 635–48 |year= 2006 |pmid= 17081983 |doi= 10.1016/j.cell.2006.09.026 }}
*{{cite journal | author=Lin D, Ippolito GC, Zong RT, ''et al.'' |title=Bright/ARID3A contributes to chromatin accessibility of the immunoglobulin heavy chain enhancer. |journal=Mol. Cancer |volume=6 |issue= |pages= 23 |year= 2007 |pmid= 17386101 |doi= 10.1186/1476-4598-6-23 }}
*{{cite journal | author=Lin D, Ippolito GC, Zong RT, ''et al.'' |title=Bright/ARID3A contributes to chromatin accessibility of the immunoglobulin heavy chain enhancer. |journal=Mol. Cancer |volume=6 |issue= |pages= 23 |year= 2007 |pmid= 17386101 |doi= 10.1186/1476-4598-6-23 | pmc=1852116 }}
}}
}}
{{refend}}
{{refend}}

Revision as of 17:33, 6 June 2010

Template:PBB AT-rich interactive domain-containing protein 3A is a protein that in humans is encoded by the ARID3A gene.[1][2]

Template:PBB Summary

Interactions

ARID3A has been shown to interact with E2F1[3] and Bruton's tyrosine kinase.[4]

References

  1. ^ Kortschak RD, Reimann H, Zimmer M, Eyre HJ, Saint R, Jenne DE (1998). "The human dead ringer/bright homolog, DRIL1: cDNA cloning, gene structure, and mapping to D19S886, a marker on 19p13.3 that is strictly linked to the Peutz-Jeghers syndrome". Genomics. 51 (2): 288–92. doi:10.1006/geno.1998.5259. PMID 9722953. {{cite journal}}: Unknown parameter |month= ignored (help)CS1 maint: multiple names: authors list (link)
  2. ^ "Entrez Gene: ARID3A AT rich interactive domain 3A (BRIGHT-like)".
  3. ^ Suzuki, M (1998). "A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer". Oncogene. 17 (7). ENGLAND: 853–65. doi:10.1038/sj.onc.1202163. ISSN 0950-9232. PMID 9780002. {{cite journal}}: Check date values in: |year= (help); Cite has empty unknown parameters: |laydate=, |laysummary=, and |laysource= (help); Unknown parameter |coauthors= ignored (|author= suggested) (help); Unknown parameter |month= ignored (help)
  4. ^ Nixon, Jamee C (2004). "The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations". Cell. Immunol. 228 (1). United States: 42–53. doi:10.1016/j.cellimm.2004.03.004. ISSN 0008-8749. PMID 15203319. {{cite journal}}: Check date values in: |year= (help); Cite has empty unknown parameters: |laydate=, |laysummary=, and |laysource= (help); Unknown parameter |coauthors= ignored (|author= suggested) (help); Unknown parameter |month= ignored (help)

Further reading

Template:PBB Further reading


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