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{{PBB_Summary
{{PBB_Summary
| section_title =
| section_title =
| summary_text = This gene encodes a member of the [[ARID domain|ARID]] (AT-rich interaction domain) family of DNA binding proteins. It was found by homology to the Drosophila dead ringer gene, which is important for normal embryogenesis. Other ARID family members have roles in embryonic patterning, cell lineage gene regulation, cell cycle control, transcriptional regulation, and possibly in chromatin structure modification.<ref name="entrez">{{cite web | title = Entrez Gene: ARID3A AT rich interactive domain 3A (BRIGHT-like)| url = http://www.ncbi.nlm.nih.gov/sites/entrez?Db=gene&Cmd=ShowDetailView&TermToSearch=1820| accessdate = }}</ref>
| summary_text = This gene encodes a member of the [[ARID domain|ARID]] (AT-rich interaction domain) family of DNA binding proteins. It was found by homology to the Drosophila dead ringer gene, which is important for normal embryogenesis. Other ARID family members have roles in embryonic patterning, cell lineage gene regulation, cell cycle control, transcriptional regulation, and possibly in chromatin structure modification.<ref name="entrez"/>
}}
}}


==Interactions==
==Interactions==
ARID3A has been shown to [[Protein-protein_interaction|interact]] with [[E2F1]]<ref name=pmid9780002>{{cite journal |last=Suzuki |first=M |authorlink= |coauthors=Okuyama S, Okamoto S, Shirasuna K, Nakajima T, Hachiya T, Nojima H, Sekiya S, Oda K |year=1998|month=Aug. |title=A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer |journal=Oncogene |volume=17 |issue=7 |pages=853–65 |publisher= |location = ENGLAND| issn = 0950-9232| pmid = 9780002 |doi = 10.1038/sj.onc.1202163 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = }}</ref> and [[Bruton's tyrosine kinase]].<ref name=pmid15203319>{{cite journal |last=Nixon |first=Jamee C |authorlink= |coauthors=Rajaiya Jaya B, Ayers Neil, Evetts Seth, Webb Carol F |year=2004|month=Mar. |title=The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations |journal=Cell. Immunol. |volume=228 |issue=1 |pages=42–53 |publisher= |location = United States| issn = 0008-8749| pmid = 15203319 |doi = 10.1016/j.cellimm.2004.03.004 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = }}</ref>
ARID3A has been shown to [[Protein-protein interaction|interact]] with [[E2F1]]<ref name=pmid9780002>{{cite journal |last=Suzuki |first=M |authorlink= |coauthors=Okuyama S, Okamoto S, Shirasuna K, Nakajima T, Hachiya T, Nojima H, Sekiya S, Oda K |year=1998|month=Aug |title=A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer |journal=Oncogene |volume=17 |issue=7 |pages=853–65 |publisher= |location = ENGLAND| issn = 0950-9232| pmid = 9780002 |doi = 10.1038/sj.onc.1202163 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = }}</ref> and [[Bruton's tyrosine kinase]].<ref name=pmid15203319>{{cite journal |last=Nixon |first=Jamee C |authorlink= |coauthors=Rajaiya Jaya B, Ayers Neil, Evetts Seth, Webb Carol F |year=2004|month=Mar |title=The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations |journal=Cell. Immunol. |volume=228 |issue=1 |pages=42–53 |publisher= |location = United States| issn = 0008-8749| pmid = 15203319 |doi = 10.1016/j.cellimm.2004.03.004 | bibcode = | oclc =| id = | url = | language = | format = | accessdate = | laysummary = | laysource = | laydate = | quote = }}</ref>


==References==
==References==
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* {{MeshName|ARID3A+protein,+human}}
* {{MeshName|ARID3A+protein,+human}}



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{{Transcription factors|g0}}
{{Transcription factors|g0}}
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[[Category:Transcription factors]]

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[[Category:Transcription factors]]


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Revision as of 14:23, 4 January 2014

Template:PBB AT-rich interactive domain-containing protein 3A is a protein that in humans is encoded by the ARID3A gene.[1][2]

Template:PBB Summary

Interactions

ARID3A has been shown to interact with E2F1[3] and Bruton's tyrosine kinase.[4]

References

  1. ^ Kortschak RD, Reimann H, Zimmer M, Eyre HJ, Saint R, Jenne DE (1998). "The human dead ringer/bright homolog, DRIL1: cDNA cloning, gene structure, and mapping to D19S886, a marker on 19p13.3 that is strictly linked to the Peutz-Jeghers syndrome". Genomics. 51 (2): 288–92. doi:10.1006/geno.1998.5259. PMID 9722953. {{cite journal}}: Unknown parameter |month= ignored (help)CS1 maint: multiple names: authors list (link)
  2. ^ "Entrez Gene: ARID3A AT rich interactive domain 3A (BRIGHT-like)".
  3. ^ Suzuki, M (1998). "A novel E2F binding protein with Myc-type HLH motif stimulates E2F-dependent transcription by forming a heterodimer". Oncogene. 17 (7). ENGLAND: 853–65. doi:10.1038/sj.onc.1202163. ISSN 0950-9232. PMID 9780002. {{cite journal}}: Cite has empty unknown parameters: |laydate=, |laysummary=, and |laysource= (help); Unknown parameter |coauthors= ignored (|author= suggested) (help); Unknown parameter |month= ignored (help)
  4. ^ Nixon, Jamee C (2004). "The transcription factor, Bright, is not expressed in all human B lymphocyte subpopulations". Cell. Immunol. 228 (1). United States: 42–53. doi:10.1016/j.cellimm.2004.03.004. ISSN 0008-8749. PMID 15203319. {{cite journal}}: Cite has empty unknown parameters: |laydate=, |laysummary=, and |laysource= (help); Unknown parameter |coauthors= ignored (|author= suggested) (help); Unknown parameter |month= ignored (help)

Further reading

Template:PBB Further reading

This article incorporates text from the United States National Library of Medicine, which is in the public domain.

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