Pages that link to "Q50663891"
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The following pages link to David L Vaux (Q50663891):
Displayed 50 items.
- Survivin and the inner centromere protein INCENP show similar cell-cycle localization and gene knockout phenotype (Q24290530) (← links)
- HtrA2 promotes cell death through its serine protease activity and its ability to antagonize inhibitor of apoptosis proteins (Q24291802) (← links)
- Mature DIABLO/Smac is produced by the IMP protease complex on the mitochondrial inner membrane (Q24299687) (← links)
- Association of mammalian sterile twenty kinases, Mst1 and Mst2, with hSalvador via C-terminal coiled-coil domains, leads to its stabilization and phosphorylation (Q24300596) (← links)
- IAP antagonists target cIAP1 to induce TNFalpha-dependent apoptosis (Q24300734) (← links)
- Asymmetric recruitment of cIAPs by TRAF2 (Q24310283) (← links)
- Inhibitor of apoptosis proteins and their relatives: IAPs and other BIRPs (Q24544173) (← links)
- The anti-apoptotic activity of XIAP is retained upon mutation of both the caspase 3- and caspase 9-interacting sites (Q24673550) (← links)
- Solution structure of a baculoviral inhibitor of apoptosis (IAP) repeat (Q27619017) (← links)
- Structure of the MDM2/MDMX RING domain heterodimer reveals dimerization is required for their ubiquitylation in trans (Q27649717) (← links)
- Structures of the cIAP2 RING domain reveal conformational changes associated with ubiquitin-conjugating enzyme (E2) recruitment (Q27652077) (← links)
- Identification of DIABLO, a mammalian protein that promotes apoptosis by binding to and antagonizing IAP proteins (Q28117928) (← links)
- Prevention of programmed cell death in Caenorhabditis elegans by human bcl-2 (Q28237519) (← links)
- The mitochondrial death squad: hardened killers or innocent bystanders? (Q28276624) (← links)
- Classification of cell death: recommendations of the Nomenclature Committee on Cell Death (Q28278883) (← links)
- Antibody production by single, hapten-specific B lymphocytes: an antigen-driven cloning system free of filler or accessory cells (Q28365857) (← links)
- The survivin-like C. elegans BIR-1 protein acts with the Aurora-like kinase AIR-2 to affect chromosomes and the spindle midzone (Q28611298) (← links)
- Entamoeba histolytica: target cells killed by trophozoites undergo DNA fragmentation which is not blocked by Bcl-2. (Q30465776) (← links)
- RIPK3 promotes cell death and NLRP3 inflammasome activation in the absence of MLKL (Q30623992) (← links)
- Identification of an Xiap-like pseudogene on mouse chromosome 7 (Q30943499) (← links)
- Direct inhibition of caspase 3 is dispensable for the anti-apoptotic activity of XIAP. (Q30992809) (← links)
- TAK1 is required for survival of mouse fibroblasts treated with TRAIL, and does so by NF-kappaB dependent induction of cFLIPL. (Q33523025) (← links)
- Signalling by CD95 and TNF receptors: not only life and death. (Q33547787) (← links)
- Prosurvival Bcl-2 family members affect autophagy only indirectly, by inhibiting Bax and Bak. (Q33767555) (← links)
- Caspase inhibitors. (Q33782509) (← links)
- Tumor necrosis factor (TNF) signaling, but not TWEAK (TNF-like weak inducer of apoptosis)-triggered cIAP1 (cellular inhibitor of apoptosis protein 1) degradation, requires cIAP1 RING dimerization and E2 binding. (Q33885155) (← links)
- Inhibition of apoptosis and clonogenic survival of cells expressing crmA variants: optimal caspase substrates are not necessarily optimal inhibitors (Q33890314) (← links)
- Two kinds of BIR-containing protein - inhibitors of apoptosis, or required for mitosis. (Q33944811) (← links)
- Apoptosis genes and autoimmunity. (Q34096282) (← links)
- Determination of cell survival by RING-mediated regulation of inhibitor of apoptosis (IAP) protein abundance (Q34132152) (← links)
- Apoptosis initiated by Bcl-2-regulated caspase activation independently of the cytochrome c/Apaf-1/caspase-9 apoptosome (Q34154044) (← links)
- Inhibitor of Apoptosis (IAP) proteins as drug targets for the treatment of cancer (Q34167279) (← links)
- Replicates and repeats--what is the difference and is it significant? A brief discussion of statistics and experimental design (Q34261739) (← links)
- Inhibition of Bak activation by VDAC2 is dependent on the Bak transmembrane anchor (Q34299265) (← links)
- Mitochondrial apoptosis is dispensable for NLRP3 inflammasome activation but non-apoptotic caspase-8 is required for inflammasome priming (Q34343275) (← links)
- A boom time for necrobiology (Q34344101) (← links)
- The caspase-8 inhibitor emricasan combines with the SMAC mimetic birinapant to induce necroptosis and treat acute myeloid leukemia (Q34527239) (← links)
- Cell death regulation by the mammalian IAP antagonist Diablo/Smac. (Q34543760) (← links)
- Error bars in experimental biology (Q34578493) (← links)
- TNFR1-dependent cell death drives inflammation in Sharpin-deficient mice. (Q34729606) (← links)
- In TNF-stimulated cells, RIPK1 promotes cell survival by stabilizing TRAF2 and cIAP1, which limits induction of non-canonical NF-kappaB and activation of caspase-8 (Q34787151) (← links)
- Apoptosis and toxicology--what relevance? (Q35035423) (← links)
- Mammalian mitochondrial IAP binding proteins (Q35120735) (← links)
- Lymphoid neoplasia and the control of haemopoietic differentiation (Q35561536) (← links)
- Alterations in the apoptotic machinery and their potential role in anticancer drug resistance (Q35567264) (← links)
- IAPs limit activation of RIP kinases by TNF receptor 1 during development (Q35874956) (← links)
- Apoptosis in the development and treatment of cancer (Q35890843) (← links)
- CrmA expression in T lymphocytes of transgenic mice inhibits CD95 (Fas/APO-1)-transduced apoptosis, but does not cause lymphadenopathy or autoimmune disease. (Q35909505) (← links)
- Toward an understanding of the molecular mechanisms of physiological cell death. (Q36073702) (← links)
- IAPs, RINGs and ubiquitylation (Q36085601) (← links)